Dementia in Football (Rugby and other contact sports)
CTE = Chronic Traumatic Encephalopathy Long Concussion Syndrome
Recovery from CTE, TBI and Concussion
Based on the Science of Neuroplasticity
Neurogenesis offers regenerative
opportunities years after injury
Reducing Chronic Symptoms of
vertigo, light sensitivity, sleep disruption,
emotional and other issues
Tools for Post-Concussion Syndrome, CTE,
Alzheimer's & Parkinson's Disease
More info on tools for therapy in CTE can be found below
Therapies Post Concussion Syndromes
Absolute diagnosis can only be achieved post-mortem, other diagnostics include
history and symptoms
sensory assessments such as VOMS
Clinic functional tests
Electro neuro diagnostics
VOMS (Subjective and Objective Tests based on thousands of Military subjects)
Innovative Computer Devices
Pupil reactions and Eye Movement. Tobii
Recovering from CTE
Post TBI Tool Box
Nutrition (IONIC first aid box)
Exercise targeted and pulsed
Sleep and Mood Management
Re-programming neural adaptation
Specialist is long term recovery
Concussion Intervention step by step
IONIC - intervene to Optmize Nerves, in TBI
Target training using Electro- neuro diagnostic tools
Accelerated and Individualized Protocols
Presentation and symptoms
Utilizing Innovative diagnostics to customized to the individual.
Mature Neuro Rehabilitation
Neural Plasticity extends for years following TBI. Improvements, if only partial recovery) can be achieve using latest developments in science.
Do not despair.
Find a Specialist
Scientific Background of repetitive brain injury in sport: Football, Rugby & other sports
Chronic traumatic encephalopathy is a degenerative brain disease caused by repetitive hitsCTE is not about single concussions. It’s the result of repeated concussions — and even head impacts that are not quite as severe — which can result in lasting structural changes in the brain. “The pain you feel [after a hit] is not necessarily an indicator of the damage that does to your head,” Philip Bayly, an engineering professor at Washington University in Saint Louis, who has been studying the mechanics of brain movement inside the head, said in a 2019 interview.Specifically, brains with CTE accumulate a protein called tau (which is believed to be dislodged from brain fibers during an injury). Tau clumps together in the tissues of the brain, interrupting critical information flow.The mechanisms of how this all happens still aren’t well understood. “The challenge is nobody sees what happens to the brain when someone gets a concussion,” Bayly said. One hypothesis is that the sulci — the grooves on the surface of the brain — experience high mechanical stress during an injury and burst open pockets of tau. (In autopsies, these clumps of tau are often found near the blood vessels at the bottom of sulci.)The disease isn’t exactly new. A form of it was originally discovered among boxers in the 1920 (who, like football players, sustain regular hits to the head). Then, it was called dementia pugilistica, or punch-drunk syndrome. Currently the only way to definitively diagnose CTE is through an autopsy. In 2005, researchers published the first confirmed case of CTE in an NFL player. The results of that report contained ominous statistics to suggest CTE might be frighteningly prevalent among players:There are approximately 0.41 concussions per NFL game of American football: 67.7% of concussions involve impact by another player’s helmet, 20.9% involve impact by other body regions (e.g., a knee), and 11.4% involve impact on the ground (29, 31, 32, 40). It has been reported that 9.3% of the concussions involved loss of consciousness and 2.4% of the concussions resulted in hospitalization. Most (92%) of the players who sustain a concussion return to practice in less than 7 days; fewer (69%) of the players who experience loss of consciousness return to practice in less than 7 days.And since then, evidence has only mounted that the repeated head blows in the NFL contribute to the disease.In 2017, Aaron Hernandez, a former player for the New England Patriots, hanged himself in prison while serving time for a violent murder. Upon an autopsy, doctors diagnosed him with CTE. It was the most severe case ever seen in a person his age (27 years old).
Symptoms of CTE include behavior
There’s a range of CTE symptoms, from forgetfulness to violent behavior.
The symptoms of CTE creep slowly, taking 8 to 10 years to manifest after initial repeated brain traumas, and can grow worse over decades. Here’s a diagram of what’s happening inside the brain as CTE grows more and more severe.
In stage I, symptoms are subtle: headaches, short-term memory loss, and loss of attention. By stage IV, “most subjects also showed profound loss of attention and concentration, executive dysfunction, language difficulties, explosively, aggressive tendencies, paranoia, depression, gait and visuospatial difficulties,” the paper explains.
Many former NFL players have CTE?
CTE can only be conclusively diagnosed in autopsy (though progress is being made in diagnosing via MRI). So it’s hard to say how many current and former football players have the condition.
What we do know is that there are dozens of cases of confirmed CTE — enough to be alarming.
In 2017, JAMA published the autopsied brains of 202 former football players, 177, or nearly 90 percent, were diagnosed with CTE.
And there was a pattern: Those who had played football longer were more likely to have worse brain damage. Among the former NFL players in the sample, 99 percent had CTE. This suggests the effects of brain trauma on CTE are cumulative. The more trauma over a longer period, the worse the symptoms.
This is a very real disease.
Football is not the only sport that poses the risk of head injury. There’s growing concern about CTE in hockey players, rugby players, Australian rules football and also Jockeys have also complained about cognitive problems after retirement
Children and School athletes suffer
Sustaining repeated head injuries during critical periods of brain maturation could alter neurodevelopmental trajectories, leading to later-life cognitive impairment
Consequences of the concussion research is that fewer young people are participating in the sport.
Athletes who began playing tackle football before age 12 developed the cognitive and behavior symptoms of CTE 13 years earlier on average than those who started playing later in their teenage years.
“Every one year younger that participants began to play football resulted in earlier reported onset of cognitive and behavioral [and] mood symptoms by approximately 2.5 years,” the study concluded. However, the study did not find an increased amount of physical abnormalities in the brains of athletes who began playing earlier than age 12.
more about youth concussion protocols>
The league has also made some steps to make the game a bit safer. Players are immediately removed from the field when there’s a potential concussion. If diagnosed, they can only return to play after completing a five-step protocol, which includes an unspecified period of rest, followed up by supervised exercise, and then examinations not just with the team doctor but also with an independent neurological consultant.
Critics, however, have charged that these protocols have been enforced unevenly across teams and players.
Furthermore, the NFL has banned helmet-to-helmet hits, made kickoff plays slightly safer, and limited the amount of contact allowed in practices. It’s also looking into artificial playing surfaces to soften the blow of impacts. Even with better helmets, football might not ever be a perfectly safe sport for brain health.
As long as football is a sport where human-to-human collisions are fundamental to play, it’s going to be a dangerous game.There’s still a lot of research to be done but we can do something today.
Eliminating the contact is not possible, so to reduce the risk with better neural protection may reduce risk, improve diagnosis, improve recovery, accelerated return to play and reduce the symptoms and deterioration in those that have been living with the issues of concussion.